Search results for "Neuroimmune system"

showing 4 items of 4 documents

Role of neuroinflammation in ethanol neurotoxicity

2019

Abstract Alcohol is a neurotoxic compound whose abuse can cause alterations in brain structure and functions, although these effects are more dramatic while the brain develops. The molecular mechanisms of neurotoxic effects of alcohol are complex and remain largely unknown. Current evidence from human and animal studies supports the role of the neuroimmune system in many actions of ethanol on the central nervous system, including neurotoxicity. Different studies have shown that, by activating innate immune receptors TLRs (Toll-like receptors) and (NOD)-like receptors (inflammasome NLRs) in glial cells, alcohol triggers signaling pathways which culminate in the production of pro-inflammatory…

Innate immune systembusiness.industryNeurotoxicityBinge drinkingAlcohol abuseInflammasomeNeuropathologymedicine.diseaseNeuroimmune systemmedicinebusinessNeuroscienceNeuroinflammationmedicine.drug
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Involvement of TLR4 in the long-term epigenetic changes, rewarding and anxiety effects induced by intermittent ethanol treatment in adolescence

2016

Studies in humans and experimental animals have demonstrated the vulnerability of the adolescent brain to actions of ethanol and the long-term consequences of binge drinking, including the behavioral and cognitive deficits that result from alcohol neurotoxicity, and increased risk to alcohol abuse and dependence. Although the mechanisms that participate in these effects are largely unknown, we have shown that ethanol by activating innate immune receptors, toll-like receptor 4 (TLR4), induces neuroinflammation, impairs myelin proteins and causes cognitive dysfunctions in adolescent mice. Since neuroimmune signaling is also involved in alcohol abuse, the aim of this study was to assess whethe…

Male0301 basic medicineEpigenetic changesmedia_common.quotation_subjectImmunologyRewarding effectsAlcohol abuseBinge drinkingAnxietyBinge DrinkingEpigenesis GeneticMice03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineRewardNeuroimmune systemmedicineAnimalsTLR4Neuroinflammationmedia_commonMice KnockoutEthanolBinge ethanol treatmentEndocrine and Autonomic SystemsAddictionAge FactorsNeurotoxicityBrainAnxiety-like behaviormedicine.diseaseEthanol preferencePrelimbic medial prefrontal cortexAdolescenceMice Inbred C57BLToll-Like Receptor 4Alcoholism030104 developmental biologySynaptic plasticityFemaleCognition DisordersPsychologyNeuroscienceMyelin Proteins030217 neurology & neurosurgeryFOSBBrain, Behavior, and Immunity
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Enterovirus and Encephalitis

2020

Enterovirus-induced infection of the central nervous system (CNS) results in acute inflammation of the brain (encephalitis) and constitutes a significant global burden to human health. These viruses are thought to be highly cytolytic, therefore normal brain function could be greatly compromised following enteroviral infection of the CNS. A further layer of complexity is added by evidence showing that some enteroviruses may establish a persistent infection within the CNS and eventually lead to pathogenesis of certain neurodegenerative disorders. Interestingly, enterovirus encephalitis is particularly common among young children, suggesting a potential causal link between the development of t…

Microbiology (medical)RNA virusvirusesencephalitisCentral nervous systemlcsh:QR1-502InflammationReviewPicornaviridaemedicine.disease_causeMicrobiologylcsh:MicrobiologyPathogenesis03 medical and health sciencesImmune systemNeuroimmune systemMedicineTropism030304 developmental biology0303 health sciences030306 microbiologybusiness.industryenterovirusvirus diseasesmedicine.diseasemedicine.anatomical_structureImmunologyEnterovirusmedicine.symptomCNSbusinessEncephalitisFrontiers in Microbiology
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Peptide neuroanatomy of adjuvant-induced arthritic inflammation in rat

1988

The influence of adjuvant-induced arthritis of the rat on central and peripheral peptide neuroanatomy was investigated by immunohistochemistry. The most striking feature of arthritic rats was the differential intensification of neuronal proenkephalin- and prodynorphin-related staining in dorsal horn. Changes were ipsilateral in monoarthritic and bilateral in polyarthritic rats as compared to controls. Opioid responsive neurons were target of substance P (SP) and calcitonin gene-related peptide (CGRP) fibers. Changes of SP and CGRP predominated in peripheral inflamed tissue and consisted of intensified immunostaining and an apparent sprouting of sensory fibers particularly around venules, in…

medicine.medical_specialtyCalcitonin Gene-Related PeptideImmunologyInflammationSubstance PSubstance PCalcitonin gene-related peptideToxicologychemistry.chemical_compoundNerve FibersNeuroimmune systemGanglia SpinalInternal medicinemedicineAnimalsPharmacology (medical)Protein PrecursorsSkinPharmacologybusiness.industryArthritisNeuropeptidesRats Inbred StrainsEnkephalinsArthritis ExperimentalImmunohistochemistryRatsProenkephalinEndocrinologyNociceptionSpinal CordchemistryCalcitoninmedicine.symptombusinessImmunostainingAgents and Actions
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